Diabetes it is a common myth that it is caused by eating too much sweets, and candies. No one really know what caused it, or at least treat it. Over the years that has bee studies on what causes it and finding a cure for it. The reasons for malfunctions that leads to the disease is yet unknown. In one medical guide put out by Better Homes and Gardens the pancreas put out insulin to help with the blood sugar levels. When the pancreas fails to put out the proper amount of insulin the blood glucose level would rise. Other causes are unknown as to why one would get diabetes.
There are two types of diabetes, commonly know by the medical community, they are Type 1 and Type 2. The reason malfunctions are unknown, but a number of predisposing and precipitating factors has been identified. There is a family history and a fault in the immune system. In the factor of the family history the risk increases for both types, indicating a possible genetic predisposition for the condition.
In Type 1 diabetes, fault in the immune system may be the factor, according to the 1998 edition of the complete reference collection. Some researchers believe that in certain genetically susceptible people, an infection of the pancreas may prompt the immune system, in effect, to turn on itself and destroy the insulin-producing beta cells. This theory is bolstered by the fact that diabetes often ensues soon after a viral infection such as the flu, chickenpox, or bad cold. Antibodies from the immune system designed to attack the beta cells of the pancreas can be found in the blood of most people destined to develop Type 1 diabetes.
In women, pregnancy may precipitate diabetes, particularly in obese women over the age of thirty who have a family history of diabetes. About half of these develop Type 1 diabetes later in life.
Because the pancreas continues to produce at least some insulin in Type 2 Diabetes, this form of disease is blames more on insulin resistance (the body is not using the hormone effectively) than on insulin deficiency or pancreatic failure. Studies have found that some Type 2 patients actually produce normal or higher than normal amounts of insulin. Studies have found patients with Type 2 diabetes may have normal insulin receptors, but this is not a universal finding, indicating that there are still other factors involved. Also, some Type 2 patients produce progressively less insulin, in time, may require insulin replacement.
Fat tissue is known to increase insulin resistance, and since people with Type 2 diabetes are often overweight there has been a tendency to attribute their diabetes to obesity. However, this now appears to be an oversimplification, and factors other than the obesity are also believed involved, although these have not been clearly identified.
Type 1 diabetes requires insulin injections coordinated with diet and exercise to control blood glucose. Most people with Type 2 diabetes do not require insulin injections (although there are exceptions); instead, most can initiall be treated by diet, exercise, and, if needed, drugs that increase the body's ability ti utilize insulin. (Later in life, however, most require insulin.
